glutathione death system enhancement for cardiac protection: pharmacological options against oxidative stress and ferroptosis Schematic representation of ferroptotic cell

The opposing effects of mTOR signaling on apoptosis may depend on the distinct pro-apoptotic stimuli

Nerve growth factor regulates CD133 function to promote tumor cell migration and invasion via activating ERK1/2 signaling in pancreatic cancer

Ferroptosis and cuproptosis: metal-dependent cell death pathways activated in response to classical chemotherapy - significance for cancer treatment

Glutathione is the most abundant cellular antioxidant in oxygen-using cells

These results suggested that the suppression of p38 MAPK phosphorylation by GHK-Cu is involved in the inhibition of the LPS-induced production of proinflammatory mediators in LPS-induced ALI