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3,5-diCQM drives melanogenesis in a dose-dependent manner, and the molecular mechanism underlying its ability to induce pigmentation is through activation of the p38 signaling pathway, phosphorylation and activation of CREB, and a cAMP/PKA dependent signaling pathway, which in turn upregulates the transcription factor MITF, thereby activating tyrosinase activity (Kim et al., 2015)

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By combining endometriosis-targeting peptide with pro-apoptotic KLAKLAK peptide23,24 and that with newly developed potent endosome-escaping HLAHLAH peptide (Fig

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They are missing an electron in their outer orbit and therefore try to steal one from a nearby healthy cell, rendering that cell dysfunctional