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On the contrary, the stable clone and wild type did not show any significant difference in their susceptibility towards doxorubicin (3 g/ml)- or radiation (0.21 Gy)-induced cell death

Reference: Hsieh et al., 2017 (VEGFR2AkteNOS signalling involvement) VEGFR2 activation triggers: VEGF binding VEGFR2 autophosphorylation (turns on VEGFR2) which leads to PI3K activation which leads to Akt phosphorylation Akt (Protein Kinase B) is critical because it phosphorylates (turns on) eNOS which leads to Nitric oxide synthesis more nitric oxide

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however, for most thiolates including that formed by glutathione, the rate of the non-enzymatic reaction is too slow to account for the inactivation of the enzymes (Forman, 2007)

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