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As we chart this terrain, it is expected to transform the post-stroke milieu from a metabolic battleground (site of competing metabolic adaptations) into a scaffold where amino acids may orchestrate metabolic repair by modulating both glycolytic flux (e.g., serine-driven one-carbon metabolism) and lipid peroxidation (e.g., cysteine-dependent glutathione synthesis), though combinatorial targeting studies are needed [186, 206]

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The second step after filtering was performed with 0.23 M Pt GSTF8 and varying dilutions of the first step reaction in buffer to cover substrate concentrations from 389 M to 6.5 M

All animal experiments were carried out under strict blinding with respect to both genotype and treatment with small molecules