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Betacarophylline, a cannabinoid receptor agonist, contributes to pain relief and nerve protection

Simultaneously, skeletal muscle in the elderly demonstrates mitochondrial dysfunction, encompassing diminished mitochondrial biogenesis, compromised oxidative phosphorylation, and an augmented production of reactive oxygen species (ROS), which further intensify endothelial oxidative damage and inflammatory responses (21, 22)

Altogether, these data indicate that FA metabolism and GSH synthesis are required to limit FA toxicity, sustaining cell proliferation and growth

These observations raise an intriguing possibility that insults in HSCs over the lifespan are a driver of maladaptive trained immunity, resulting in aging-associated inflammation and functional decline of distant tissues

Antioxid Redox Signal, 2018