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Second, informative injury models need to mimic the injuries seen in sporting environments (Wall and Shani, 2008), yet most brain injury models create focal hemorrhagic cortical lesions caused by direct impact to the skull or exposed brain (Xiong et al., 2013), while the human concussed brain is more commonly associated with distributed white matter alterations (Benson et al., 2007

Palmitoylation of cysteine residues located at the N-terminus of GAD65 is required for the transport of GAD65 to synaptic terminals [51], and decreased palmitoylation impairs GABAergic neurotransmission, as observed in Huntingtons disease [52]