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Available reports have demonstrated that the increase of HDACs activity triggers protein deacetylation, leading to the pathogenesis of a plethora of human diseases including epilepsy [37, 38] and memory deficits [39]

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Data supporting the conclusions of this article are included within the article

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The investigators concluded that decreased hepatic GSH concentrations in patients with liver disease may represent a contributing factor of liver injury susceptibility and toxicity risk in these patients