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Animal studies further indicate parenchymal accumulation and microglial activation, where intracellular 50500 nm particles tend to correlate with cytotoxic effects ( 3.2 Oxidative stress and neuroinflammation Oxidative stress is a central mechanism in MPs-induced neurotoxicity

More severe deficiencies result in fatal encephalopathy and multiple systems atrophy

GilCampos, M

To determine whether these transcriptomic changes were reflected at the metabolic level, we quantified -hydroxybutyrate, a terminal product of -oxidation, in both cell lysates and culture supernatants

For instance, compounds like kojic acid show promising inhibitory effects but face limitations due to cytotoxicity or allergic reactions