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Acknowledgements The authors would like to thank the experimental apparatus and technical support provided by the Key Laboratory for Metabolic Diseases in Chinese Medicine, Nanjing University of Chinese Medicine

Damaged mitochondria produce abnormally high levels of ROS [89], which contribute to the deterioration of cognitive function and interfere with normal cellular processes in neurons [78,90]

Some patients experience significant progress, and their traits may become less noticeable or no longer meet full diagnostic criteria

This study delves into the mechanisms underlying breast cancer and ROS-related cell death, along with the associated epigenetic regulatory network, and systematically analyses the impact of different PCD modalities on tumour evolution and treatment, shedding new light on the optimisation of clinical treatment strategies and the development of emerging nanoparticle-targeted cell death therapies

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