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We recognize that distinguishing cadmium-specific effects from effects caused by PAHs, TSNAs, and other volatile compounds would require additional biomonitoring (e.g., blood and urinary cadmium levels, urinary PAH metabolites), mechanistic biomarkers (e.g., metallothionein, 2-microglobulin, 8-hydroxydeoxyguanosine), and DNA adduct analysis

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