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A crosstalk between apoptosis and autophagy exists, and the interaction of anti-apoptotic regulators such as Bcl-2 and Bcl-xl suppresses the autophagy promoter Beclin-1 activity, resulting in autophagy inhibition (Pattingre et al., 2005)

This process, known as non-enzymatic lipid peroxidation, is further fueled by Fe 2+ -dependent fenton reactions, which convert hydrogen peroxide (H 2 O 2 ) to OH and oxidize L-OOH by cleaving its O-O bond to generate LO and hydroxide ions (OH )

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The more friction you pile on, the easier it is for the gut to overreact