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At the same time, accumulated research on EndMT showed that endothelial dysfunction contributes to the pathogenesis of pulmonary hypertension [132] and pulmonary vascular endothelial cell apoptosis can promote the development of pulmonary hypertension, which is a common complication of COPD and is closely related to COPD progression [133]

The results of the CCK-8 assay revealed that CSE at concentrations ranging from 0.251% had no significant effect on cell viability after 24 h of treatment (data not shown)

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Neuroscience 164 (2), 798808 (2009)