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Through transcriptional activation of target genes governing cell cycle arrest, DNA repair, senescence, and apoptosis, P53 coordinates adaptive responses that mobilize autophagy and ferroptosis mechanisms and promotes the elimination of compromised cells while suppressing carcinogenesis (Duffy et al

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These findings suggest that by suppressing the activation of NF-B and MAPKs, colchicine inhibited senescence in endothelial cells treated with tobacco smoke condensate

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