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As an analog of endogenous amylin, it is studied for its receptor-mediated signaling dynamics, sustained activation profile, and downstream neuroendocrine cascades within controlled laboratory environments

Hepatic dysfunction critically contributes to metabolic syndrome, non-alcoholic fatty liver disease (NAFLD) and fibrotic progression (13,15)

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The GABAergic Deficit Hypothesis of Major Depressive Disorder. Molecular Psychiatry, vol