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Notably, they exhibited lesser interstitial edema (only mild), markedly less damaged neurons than corresponding controls and had no inflammatory reaction ( Table 8 )

Most of the beneficial effect of semaglutide on UACR reduction was mediated by HbA1c, BW and BP reduction 79

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We then discuss the favorable effect of HGF in promoting neuron survival in the context of brain injury and in neurodegenerative disorders, pointing out its pro-angiogenic, anti-fibrotic, anti-inflammatory, immune-modulatory, and neuro-regenerative properties

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