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in parallel, bacterial killing assays of the same strain were performed by the same medium adjusted to the equivalent pH levels with HCl (pH

The lack of GSH detection can be explained by the possibility that free GSH is maintained in apoplast at a minimum level and is transported back to cytosol after GSSG is reduced

Anti-apoptotic protection of dermal papilla cells AHK-Cu protected DPCs from programmed cell death through multiple molecular pathways: Bcl-2/Bax rebalancing: Bcl-2 (an anti-apoptotic protein dominant during the telogen-to-anagen transition) was upregulated, while Bax (a pro-apoptotic protein) was downregulated

It is unclear what biological processes regulate such alteration