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These results indicate that the OXA-induced increase in liver free radicals, massive depletion of SOD and the insufficient compensation of GSH-Px and GSH syntheses all lead to the occurrence of ALI

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AMPK also inhibits lipid biosynthesis by suppressing transcription factor sterol regulatory element-binding protein 1 (SREBP1) and promoting the action of peroxisome proliferator-activated receptor alpha (PPAR), a nuclear receptor that acts to enhance both mitochondrial and peroxisomal FA -oxidation 23