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G6PD deficiency has been identified as a major risk factor in severe neonatal hyperbilirubinemia that may lead to the debilitating complication of kernicterus

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Il agit comme cofacteur pour certaines enzymes impliques dans : la dtoxification la neutralisation des toxines la rgulation du mtabolisme cellulaire Il est notamment essentiel au bon fonctionnement des enzymes de la famille des glutathion peroxydases , qui jouent un rle cl dans la protection contre les peroxydes

Similarities between precursors of AGEs and ALEs and haptens pose several interesting questions: would haptenation change the profiles of AGEs and ALEs and alter the downstream signaling or recognition