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Lastly, genetic manipulation of antioxidant genes, such as Sod1 , Gpx 1 and Cat , in animal models alters susceptibility to alcohol-induced liver injury 28,29,30

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Given the essential role of SLC25A39 in mitochondrial glutathione transport, our results provide a mechanistic explanation for how SLC25A39 loss causes mitochondrial defects, neurodegeneration and anemia in yeast, flies and mice, respectively