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It does so through an epigenetic mechanism, where FGF15/19 interacts with its target nuclear receptors, such as small heterodimer partner (SHP), which then recruits DNA methyltransferase-3a (DNMT3a) to the lipogenic gene loci, including FASN

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Once the S-A-GSH molecule is safely inside the target cell, specialized enzymes called intracellular thioesterases recognize the molecule and cleave (remove) the acetyl group

Maintaining GSH levels could slow age-related neurodegeneration.Acute Brain Injuries:In conditions like stroke or traumatic brain injury, protecting mitochondrial GSH may reduce neuronal damage and improve recovery