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& Melton, D

It is believed that by modulating the tumor microenvironment (TME) post-treatment, not only may the efficacy of current treatments be improved, but such associated negative side effects, as well as further complications arising from treatment, including metastasis, have the potential to be reduced

This process begins with endothelial dysfunction, a critical event in the pathogenesis of atherosclerosis [64]

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We therefore hypothesize that SFB may have both co-evolved with its host and, at times, crossed over into different hosts, potentially through sharing of the same environment