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B cells are also central to the pathogenesis of RA, contributing to autoantibody production, inflammation, and immune dysregulation, and B cells depletion with anti-CD20 antibody significantly improve RA symptoms (Roll et al., 2006) (Figure 1)

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A comparative analysis of two pivotal studies highlights key parameters influencing therapeutic outcomes

Without sufficient selenium, the effectiveness of glutathione could be compromised, leading to reduced antioxidant defense