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Mechanistically, myristoylation promotes the recruitment of FSP1 to the plasma membrane, where it functions as an oxidoreductase, mediates the reduction of NADH-dependent CoQ, and produces a lipophilic free radical-trapping antioxidant (RTA) that inhibits the lipid peroxidation chain reaction [6, 11, 88]

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In the MASLD (metabolic dysfunction-associated steatotic liver disease) trial, retatrutide reduced liver fat by up to 82.4% at the 12mg dose level

In fact, mTORC2 is known to activate serum/glucocorticoid regulated kinase 1 (SGK1), that will then stimulate ENaC and NHE-3, increasing sodium reabsorption (Figure 4B)

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