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Isolating the effect of specific blood factors on microglia activity is difficult and likely complex

A peptides, in particular, have been shown to disrupt mitochondrial function by interacting with mitochondrial membranes and promoting the production of ROS [156]

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LONP1 in human diseases As a central regulator of mitochondrial PQC, LONP1 dysregulation contributes to the pathogenesis of diverse human diseases, including neurological, cardiovascular, pulmonary, digestive, renal, reproductive, and musculoskeletal system disorders, and malignant tumors (Fig