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The copper ionophore elesclomol exhibits a paradoxical dual role that is critically dependent on the cellular context

In addition to excessive intake of dietary supplements, parenteral administration of vitamin B12 may be another cause of excess, which is likely associated with an immune system response to produce anti-TC II antibodies

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Egr-1 induces the expression of its corepressor nab2 by activation of the nab2 promoter thereby establishing a negative feedback loop

Thus, antioxidant therapies must take into consideration this delicate redox balance and ensure that basal levels of reactive species are maintained for proper cellular functioning