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This modification enhances its biological activity, resulting in a higher affinity for the IGF-1 receptor and a lower affinity for IGF-binding proteins compared to endogenous IGF-1

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Hypoxia inhibits ferritinophagy, increases mitochondrial ferritin, and protects from ferroptosis

Its biocompatibility, low cytotoxicity, and PEG-like stealth properties, combined with enhanced stability against hydrolysis and reduced crystallinity, make PEtOx ideal for drug delivery, tissue engineering, hydrogels, micelles, nanoparticles, and antifouling coatings [79]

Once aggregated, those molecules are permanently denatured