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Researchers and scientists identified BPC-157 during gastric mucosal/gastric juice studies and decided to later develop a synthetic version to study it
The insulin resistance concern disqualifies it from metabolic research in anyone over 50 or with BMI above 27
Key contributors to atherogenesis include oxidative stress (excess ROS oxidizing lipids and damaging endothelial cells), vascular inflammation (cytokine and chemokine release recruiting more immune cells), endothelial dysfunction (reduced NO bioavailability and loss of barrier integrity), and dyslipidemia (elevated LDL/triglycerides and low HDL)
The 22.7% result exceeded what simple addition would predict (11.8 + 16.1 = 27.9, minus baseline placebo effect)
My Clinical Take I understand why BPC-157 is appealing
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