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Chronic low-grade inflammation in adipose tissue is a critical driver of MS, and GPER activation attenuates this inflammation, promoting fatty acid oxidation and energy metabolism [133, 175]

We observed the dissociation of p53 TAD (13-61) from FOXO4 FHD upon the addition of p53 TAD PEP, resulting in a chemical shift of the 15 N-p53 TAD-FOXO4 FHD complex towards the unbound state of p53 TAD (Figure