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In particular, it highlights the role of the thiomethyl shunt pathway to form thiomethyl metabolites that could be used in the future for a better typing of patients related to the kinetic elimination of APAP and to assess inter-individual susceptibility to APAP-induced hepatotoxicity

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This therapy helps reduce systemic inflammation which is a primary driver of chronic discomfort

The main reason: the lack of randomized controlled clinical trials in humans