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Aggarwal et al., 2016

Nicotinamide N-methyltransferase inhibition mimics and boosts exercise-mediated improvements in muscle function in aged mice

Many mechanisms have been implicated in heat stress nephropathy, involving primarily heat-induced inflammation, reduced renal blood flow in the face of increased Na + K + ATPase activity (supplydemand mismatch), dehydration, and hyperosmolarity-induced activation of polyol fructokinase pathway that results in hyperuricemia and vasoconstriction, and subclinical muscle injury, releasing muscle creatine due to exercise

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Journal of Biochemical and Molecular Toxicology