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Our study showed that HepG2 cells, which primarily rely on extracellular cystine as a source of cysteine 20 , responded to cystine deprivation by reducing anabolic signaling including attenuation of mTORC1 pathway and activation of the ISR (Fig

"An electron paramagnetic resonance study of 3,4-dihydroxyphenylethylamine beta-hydroxylase"

JBT, JZ, and SEW received grants from the NIH, including 1 R01 AI145406-01A1, 5 P01 AI106684, and U10 HL109152, and donations from the Dellenback Fund