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MOTS-c has a short half-life (46 hours), so missing a single dose doesn't create long-term metabolic disruption, but it does interrupt the AMPK activation pattern for that 48-hour window

Because it acts at the cellular level rather than on appetite or hormones, interest in it extends beyond weight loss into longevity and metabolic disease prevention

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Further, it activates muscle stem cells, promoting tissue regeneration, and reverses insulin resistance in relevant animal models, all with good systemic tolerability 1,2

It provides sufficient material for multiple experimental runs while minimizing waste and supporting precise measurement