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The persistent hyperglycaemic state of diabetes leads to the deposition of advanced glycosylation end-products (AEGs) in the vascular wall of diabetic patients

Failure of intravenous N -acetylcysteine to reduce methemoglobin produced by sodium nitrite in human volunteers: a randomized controlled trial

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This mechanism of action also explains why the body contains such large stores of reduced glutathione and why topping off these glutathione stores with supplemented reduced glutathione can help promote ideal oxidative status throughout the body