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Hepatic PRLR gene expression was significantly reduced in patients with NAFLD and negatively correlated with CD36 gene expression, a key transporter of free fatty acid uptake in the liver involved in NAFLD development

Ahmad, O., Wang, B., Ma, K., Deng, Y., Li, M., & Yang, L

revealed that F-53B triggers activation of the LOC101929922/miR-542-3p/ACSL4 signaling axis, thereby inducing ferroptosis and phenotypic switching in VSMCs, which contributs to vascular remodeling

This metabolic milieu generates reactive oxygen species (ROS) that can damage photoreceptor cells, retinal pigment epithelium, and choroidal vasculature

The eligibility requirement is the same as for brand-name drugs: a valid prescription tied to a documented diagnosis