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These findings suggest that GHK-Cus anti-inflammatory action is mediated, at least in part, by modulating the p38 MAPK and NF-B signaling pathways [1]
Fast liquid chromatography (FPLC) analysis of nephrotic serum revealed a decreased 150 kDA IGFBP ternary complex due to reduced intact IGFBP-3, whereas IGFBP-3 low molecular weight fragments were markedly increased, suggesting increased proteolytic degradation of IGFBP-3
It can assist in precise cancer diagnosis, targeted drug delivery, and improved therapeutic efficacy [108, 109]
