Collectively, these data validated the hypothesis that DR-EVs facilitate drug resistance and that the resistance to PTX could be transmitted from drug-resistant cells to sensitive cells
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This is consistent with the passage of microbial substances to the systemic circulation and the activation of inflammatory pathways
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Indeed, for ILC2s we observed that this concentration did not precipitate changes in cellular viability ( Figures 3F, G ), and was additionally required to produce maximal suppression of ILC2 activity ( Figures 3H, I )
