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While classical gene mutations are region-limited, epimutations often occur early in cancer development and have a genome-wide impact, boosting on the one hand the expression of cell survival genes or proto-oncogenes and deactivating on the other hand tumor suppressor genes (TSGs), DNA repair mechanisms as well as cell division brakes, thus leading to carcinogenesis (Esteller, 2008

Parallel ultrastructural analysis of normal human fibroblasts showed a typical spindle-like shape and a cytoplasm with a few rough endoplasmic reticulum cisternae and intracellular filaments (Fig

We quantitatively frame its hormetic behavior (a biphasic dose-response phenomenon characterized by low-dose stimulation and high-dose inhibition) and mechanistically explain its synergistic potential with conventional therapies

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