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These observations demonstrate that GSH and GSH-rich YE offer a defense against liver damage in ethanol-induced oxidative stress, primarily by enhancing the expression of antioxidant enzymes via the activation of the Nrf2/Keap1 signaling pathway

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In the tumor microenvironment, oxidative stress promotes the expression of VEGF and tumor angiogenesis, not only providing essential oxygen and nutrients for cancer cells but also involved in removing metabolic wastes [146]

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Of the above-mentioned ROIs, GSH levels in the frontal (Hupfeld et al., 2021, Tong et al., 2016) and occipital (Emir et al., 2011, Tong et al., 2016) regions were assessed by both MRS and biochemical analyses