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Taken together with the earlier investigation in SAPAP3 knockouts (Welch et al., 2007), these effects of Slitrk5 knockout show how distinct genetic perturbations can lead to different abnormalities in CSTC glutamatergic neurotransmission, and yet to similar and interesting behavioral phenotypes

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Golding, M

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Induction of hemolysis and eryptosis by occupational pollutant nickel chloride is mediated through calcium influx and p38 MAP kinase signaling

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Chromosomal Aberrations Predict Malignant Progression of Esophageal Adenocarcinoma: a Genome-wide High-Density SNP Array Analysis

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