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The implications of NNMT inhibition extend far beyond simple cofactor availability, influencing fundamental cellular processes including mitochondrial biogenesis, oxidative stress management, and epigenetic regulation

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The presence of previous neurological damage from B12 deficiency may necessitate more frequent injections to prevent symptom recurrence, even after initial improvement

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Neurexins transsynaptic complexes play a central and important role in synaptic plasticity [26, 28], and the role of synaptic plasticity in sustained antidepressant effect has been widely reported [11, 16, 25]