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Unlike other CNS disorders, cerebral ischemias lactylation dysregulation is uniquely driven by acute hypoxia-induced mitochondrial dysfunction and ANLS collapsekey pathogenic targets include LCP1 and ARF1 lactylation, which specifically mediate calcium overload and mitochondrial transfer impairment

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Possessing stem-like properties, tumor-repopulating cells (TRCs) develop therapeutic resistance to both chemotherapy and irradiation through mechanisms that reduce their susceptibility to ferroptotic cell death (81)