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Epithelial-mesenchymal transition links inflammation and fibrosis in the pathogenesis of endometriosis: a narrative review

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It has been shown that glioma cells overexpressing galectin-1 are capable to evade NK immune surveillance, whereas the lack of galectin-1 leads to accentuated tumor-killing effects of NK cells by releasing granzyme B [50, 77]

According to the Mounjaro Summary of Product Characteristics (SmPC), these two treatments do not have a known direct pharmacological interaction, as they work through entirely different mechanisms and are metabolised via separate pathways