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In the academic discourse, the prevailing research delineates DILI into two principal subclasses: intrinsic hepatotoxicity and idiosyncratic hepatotoxicity

Hcy can either enter the transsulfuration pathway or be remethylated back to methionine via the methionine synthase (MS) pathway or the betaine-homocysteine S-methyltransferase (BHMT) pathway, thereby completing the methionine cycle 41,43,44 (Fig

As system Xc - is critical to provide cysteine for the biosynthesis of GSH [3], which is required for GPX4 activity [19], we hypothesized that ferroptosis susceptibility upon direct inhibition of GPX4 should not be affected by dexamethasone

doi: 10.1016/j.therap.2020.06.012 13 DanserA

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