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When GFAT is overexpressed in mice under the control of a GLUT4 promoter the animals develop classical insulin-resistant phenotype with hyperinsulinemia and reduction in whole-body glucose disposal rate

Bcl-xL-inhibitory BH3 mimetics can induce a transient thrombocytopathy that undermines the hemostatic function of platelets

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The same paper is refreshingly candid about the gaps: key questions like ideal treatment duration, how long any effect lasts, and maintenance dosing were still unanswered as of publication