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One possible mechanism for the antineoplastic effect is the inhibition of IL-6 signalling pathways, which suppresses cellular invasion, and another is the abrogation of cell cycle progression in breast cancer cells (specifically MDA-MB-231 cells) via the down-regulation of important cyclins and CDK1 (160)

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The related damage can result in implantation failure, recurrent pregnancy loss, preterm labor and/or placenta accreta [78]

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