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Nature (2008) 453(7191):6571

Considering this, CVB infection, particularly persistent infection, may contribute to the overall demise of the cell population by further activating autoreactive T cells, augmenting the effects of autoimmunity, and by further inducing cell stress and death

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BPC-157 promotes angiogenesis through interaction with vascular endothelial growth factor (VEGF) and the nitric oxide system, effectively building new supply lines to deliver oxygen and nutrients to damaged areas