The extracellular citrulline autoantigen present in the joints of RA patients is mainly derived from the increased NET, which can cause the inflammatory response of synovial fibroblasts in patients and release a series of inflammatory mediators (such as pro-inflammatory cytokines, adhesion molecules and chemokines) to drive the progression of RA (205)
The anti-inflammatory effects were assessed by evaluating the permeability of the prepared microneedle gel
This reduction in glutathione levels is associated with increased susceptibility to oxidative stress and age-related health issues
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J Histochem Cytochem
