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Figure 6 This observation leads us to conclude that vitamin B 12 deficiency must have a secondary effect of inhibiting CBS activity, which in turn inhibits the flux of Hcy converting into cystathionine and protects GSH from changes in Hcy

Whether injected, oral, or intranasal GHK-Cu would replicate the gene expression changes seen in cell culture studies in living humans is unproven

Intracellular replication has been found to be supported by TonB-dependent iron acquisition from a host source, such as ferritin, the degradation of which is promoted by the pathogen (Larson et al., 2002, Larson et al., 2004)

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