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In neurodegenerative disease, the histone acetylation homeostasis is significantly compromised, shifting towards hypoacetylation (Saha and Pahan, 2005)

Factors involved in induction and progression of autophagy, especially AMPK, TFEB, LC3 and BECN1, play crucial roles in mitophagy (Reference Wang, Chen and Li48, Reference Zheng, Wang and Zhou49)

This leads to its translocation into the nucleus and subsequent stimulation of various genes responsible for antioxidant activity, such as NAD(P)H quinone oxidoreductase 1 (NQO1), heme oxygenase 1 (HO-1) and glutathione peroxidase (13)

This discrepancy is not unique to antioxidants

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