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Its pharmacological signature potent GHS-R1a-mediated GH release with negligible off-target stimulation of cortisol, prolactin, or ACTH at research-relevant concentrations makes it the ghrelin receptor agonist of choice for investigators requiring isolated GHS-pathway activation without confounding neuroendocrine variables

GHRP-2 and GHRP-6 produce measurable increases in these hormones

Cleavage of structural proteins during the assembly of the head of bacteriophage T4